TBS Scholars
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Stephanie Davis |
Name:
Stephanie Davis
Title: MD, PhD candidate (Georgetown University)
Email: sed51@georgetown.edu
Research Topic: Molecular mechanisms of multiple sclerosis
Mentors: Jeffery Huang, PhD (Department of Biology, Georgetown University) & Anton Wellstein MD, PhD (Department of Oncology, Georgetown University)
Pubmed: See link
Bio: As a third year MD/PhD candidate at Georgetown University, I am interested in understanding the mechanisms of myelin repair in multiple sclerosis, with the ultimate goal of contributing to therapeutic advancement. I was first introduced to neuroscience research during my undergraduate degree at Barnard College, Columbia University, in the laboratory of Dr. Elizabeth Bauer. There I studied the molecular and behavioral mechanism of fear learning and fear extinction in Sprague-Dawley rats. I learned to perform stereotaxic surgery for the implantation of cannulae into the basolateral amygdala, to design and run behavioral protocols, to analyze behavioral data and to perform histological analysis of brain slices. Specifically, I studied the effects of L-type voltage-gated calcium channel blockade on fear extinction. My work in this lab led to a first author publication in the Journal of Neuroscience the following year. After graduating Cum Laude from Barnard College, I spent 6 months as a paid intern in the Laboratory of Neuronal Microcircuitry headed by Dr. Henry Markram of the Blue Brain Project at the École Polytechnique Fédérale de Lausanne, Switzerland, where I studied the effects of various splice variants of transient receptor potential cation channels in layer 5 pyramidal neurons in mice. In 2012, I began my career as an MD/PhD candidate at Georgetown University. Clinical, personal, academic, and research experiences during my first two years in the MD/PhD program have drawn my attention to multiple sclerosis. I am currently conducting my PhD thesis in the lab of Dr. Jeffrey Huang, investigating the roles of inflammation and central nervous system remyelination in mouse models of multiple sclerosis.
Title: MD, PhD candidate (Georgetown University)
Email: sed51@georgetown.edu
Research Topic: Molecular mechanisms of multiple sclerosis
Mentors: Jeffery Huang, PhD (Department of Biology, Georgetown University) & Anton Wellstein MD, PhD (Department of Oncology, Georgetown University)
Pubmed: See link
Bio: As a third year MD/PhD candidate at Georgetown University, I am interested in understanding the mechanisms of myelin repair in multiple sclerosis, with the ultimate goal of contributing to therapeutic advancement. I was first introduced to neuroscience research during my undergraduate degree at Barnard College, Columbia University, in the laboratory of Dr. Elizabeth Bauer. There I studied the molecular and behavioral mechanism of fear learning and fear extinction in Sprague-Dawley rats. I learned to perform stereotaxic surgery for the implantation of cannulae into the basolateral amygdala, to design and run behavioral protocols, to analyze behavioral data and to perform histological analysis of brain slices. Specifically, I studied the effects of L-type voltage-gated calcium channel blockade on fear extinction. My work in this lab led to a first author publication in the Journal of Neuroscience the following year. After graduating Cum Laude from Barnard College, I spent 6 months as a paid intern in the Laboratory of Neuronal Microcircuitry headed by Dr. Henry Markram of the Blue Brain Project at the École Polytechnique Fédérale de Lausanne, Switzerland, where I studied the effects of various splice variants of transient receptor potential cation channels in layer 5 pyramidal neurons in mice. In 2012, I began my career as an MD/PhD candidate at Georgetown University. Clinical, personal, academic, and research experiences during my first two years in the MD/PhD program have drawn my attention to multiple sclerosis. I am currently conducting my PhD thesis in the lab of Dr. Jeffrey Huang, investigating the roles of inflammation and central nervous system remyelination in mouse models of multiple sclerosis.